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De Kouchkovsky, I. Blood Cancer J. Coombs, C. Acute promyelocytic leukemia: where did we start, where are we now, and the future. Lo-Coco, F. Retinoic acid and arsenic trioxide for acute promyelocytic leukemia. Reprogramming acute myeloid leukemia into sensitivity for retinoic-acid-driven differentiation. Breitman, T. Induction of differentiation of the human promyelocytic leukemia cell line HL by retinoic acid.

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Xu, Q. Survival of acute myeloid leukemia cells requires PI3 kinase activation. Blood , , 3 , Min, Y. Leukemia , , 17 5 , Kubota, Y. Constitutive activation of PI3K is involved in the spontaneous proliferation of primary acute myeloid leukemia cells: direct evidence of PI3K activation. Leukemia , , 18 8 , Grandage, V. Leukemia , , 19 4 , Antileukemic activity of rapamycin in acute myeloid leukemia. Blood , , 6 , Dinner, S. Targeting mTOR pathway in leukemia. Tabe, Y. Inhibition of mTOR kinase as a therapeutic target for acute myeloid leukemia.

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Ghosh, J. S6K1 regulates hematopoietic stem cell self-renewal and leukemia maintenance. Gao, Y. Food and Drug Administration. Li, J. Rapamycin: one drug, many effects. Pollizzi, K. Regulation of T cells by mTOR: the known knowns and the known unknowns. Trends Immunol. Fantus, D. Evolving perspectives of mTOR complexes in immunity and transplantation. Eng, C.

Activity of rapamycin AY, against transplanted tumors. Tokyo , , 37 10 , Price, D. Rapamycin-induced inhibition of the kilodalton S6 protein kinase. Muthukkumar, S. Rapamycin, a potent immunosuppressive drug, causes programmed cell death in B lymphoma cells. Transplantation , , 60 3 , Brown, V. Rapamycin is active against B-precursor leukemia in vitro and in vivo, an effect that is modulated by ILmediated signaling.

USA , , 25 , Avellino, R. Rapamycin stimulates apoptosis of childhood acute lymphoblastic leukemia cells. Mohi, M. Combination of rapamycin and protein tyrosine kinase PTK inhibitors for the treatment of leukemias caused by oncogenic PTKs. USA , , 9 , Blood , , 13 , Rizzieri, D.

A phase 2 clinical trial of deforolimus AP, MK , a novel mammalian target of rapamycin inhibitor, in patients with relapsed or refractory hematologic malignancies. Yee, K. Perl, A. A phase I study of the mammalian target of rapamycin inhibitor sirolimus and MEC chemotherapy in relapsed and refractory acute myelogenous leukemia.

Amadori, S. Park, S. Leukemia , , 27 7 , Calimeri, T. Bertagnolo, V. Phosphoinositide 3-kinase activity is essential for all-trans-retinoic acid-induced granulocytic differentiation of HL cells. Sykes, S. Cell , , 5 , Jayaraman, T. Rapamycin-FKBP12 blocks proliferation, induces differentiation, and inhibits cdc2 kinase activity in a myogenic cell line. Ogawa, T. Yamamoto-Yamaguchi, Y.

Blockade of mTOR signaling potentiates the ability of histone deacetylase inhibitor to induce growth arrest and differentiation of acute myelogenous leukemia cells. Leukemia , , 22 12 , Gadhoum, S. Leukemia , , 30 12 , Yang, J.

Dembitz, V. The mechanism of synergistic effects of arsenic trioxide and rapamycin in acute myeloid leukemia cell lines lacking typical t 15;17 translocation. Isakson, P. Neri, L. Monocytic differentiation of HL cells is characterized by the nuclear translocation of phosphatidylinositol 3-kinase and of definite phosphatidylinositol-specific phospholipase C isoforms.

Zhang, Y. AKT pathway is activated by 1, dihydroxyvitamin D3 and participates in its anti-apoptotic effect and cell cycle control in differentiating HL60 cells. Cell Cycle , , 5 4 , Carling, D. Purification and characterization of the AMP-activated protein kinase. Copurification of acetyl-CoA carboxylase kinase and 3-hydroxymethylglutaryl-CoA reductase kinase activities. AMPK signalling in health and disease.

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