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Xu, Q. Survival of acute myeloid leukemia cells requires PI3 kinase activation. Blood , , 3 , Min, Y. Leukemia , , 17 5 , Kubota, Y. Constitutive activation of PI3K is involved in the spontaneous proliferation of primary acute myeloid leukemia cells: direct evidence of PI3K activation. Leukemia , , 18 8 , Grandage, V. Leukemia , , 19 4 , Antileukemic activity of rapamycin in acute myeloid leukemia. Blood , , 6 , Dinner, S. Targeting mTOR pathway in leukemia. Tabe, Y. Inhibition of mTOR kinase as a therapeutic target for acute myeloid leukemia.

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Activity of rapamycin AY, against transplanted tumors. Tokyo , , 37 10 , Price, D. Rapamycin-induced inhibition of the kilodalton S6 protein kinase. Muthukkumar, S. Rapamycin, a potent immunosuppressive drug, causes programmed cell death in B lymphoma cells. Transplantation , , 60 3 , Brown, V. Rapamycin is active against B-precursor leukemia in vitro and in vivo, an effect that is modulated by ILmediated signaling.

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Blockade of mTOR signaling potentiates the ability of histone deacetylase inhibitor to induce growth arrest and differentiation of acute myelogenous leukemia cells. Leukemia , , 22 12 , Gadhoum, S. Leukemia , , 30 12 , Yang, J.

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